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American Society of Microbiology ABMM Sample Questions (Q162-Q167):

NEW QUESTION # 162
A research team is investigating the mechanism of bacterial resistance to a novel fluoroquinolone antibiotic.
They isolate a resistant strain of Escherichia coli from a patient with a urinary tract infection. Genetic analysis of this strain reveals a point mutation in the gyrA gene. This mutation MOST likely results in:

A. Increased expression of efflux pumps that actively transport the antibiotic out of the cell.
B. Production of an enzyme that inactivates the fluoroquinolone molecule.
C. Alteration of the DNA gyrase enzyme, reducing its affinity for the fluoroquinolone.
D. Modification of the ribosomal target site, preventing antibiotic binding.

Answer: C

NEW QUESTION # 163
A patient with HIV infection and a CD4+ T-cell count below 100 cells/µL presents with headache, fever, and altered mental status. CSF analysis reveals elevated protein and low glucose. India ink stain is negative.
Cryptococcal antigen test of the CSF is positive. The MOST appropriate initial treatment is:

A. Fluconazole
B. Amphotericin B and flucytosine
C. Caspofungin
D. Voriconazole

Answer: B

NEW QUESTION # 164
A public health investigation is underway following several cases of encephalitis in a specific geographic region during the summer months. Mosquitoes are abundant in the area. Serological testing of patient CSF samples reveals IgM antibodies against a flavivirus. The MOST likely causative agent is:

A. Jamestown Canyon virus
B. West Nile virus
C. Eastern equine encephalitis virus
D. Rabies virus

Answer: B

NEW QUESTION # 165
A neonate develops a vesicular rash, fever, and seizures within the first week of life. CSF analysis reveals lymphocytic pleocytosis and elevated protein. PCR of the CSF is positive for a double-stranded DNA virus.
The MOST likely causative agent is:

A. Herpes simplex virus (HSV)
B. Human parvovirus B19
C. Varicella-zoster virus (VZV)
D. Cytomegalovirus (CMV)

Answer: A

NEW QUESTION # 166
A research laboratory is investigating the mechanism of action of a novel antiviral compound. In vitro studies demonstrate that the compound effectively inhibits the replication of a specific enveloped RNA virus. Further analysis reveals that the compound does not interfere with viral entry, genome replication, or protein synthesis. However, electron microscopy of infected cells treated with the compound shows an accumulation of viral glycoproteins within the endoplasmic reticulum and Golgi apparatus, with a significant reduction in the release of infectious virions. The MOST likely target of this antiviral compound is:

A. Host cell membrane fusion machinery
B. Viral glycoprotein trafficking and maturation
C. Viral RNA-dependent RNA polymerase
D. Viral protease responsible for polyprotein cleavage

Answer: B

NEW QUESTION # 167
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